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Endoplasmic reticulum stress is activated after epibrassinolide treatment leading autophagy in wild type and Atg5-/- mouse embryonic fibroblasts: a survival mechanism in nonmalignant cells
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K. Adacan Et Al. , "Endoplasmic reticulum stress is activated after epibrassinolide treatment leading autophagy in wild type and Atg5-/- mouse embryonic fibroblasts: a survival mechanism in nonmalignant cells," 42nd Congress of the Federation-of-European-Biochemical-Societies (FEBS) on From Molecules to Cells and Back , Jerusalem, Israel, pp.307, 2017

Adacan, K. Et Al. 2017. Endoplasmic reticulum stress is activated after epibrassinolide treatment leading autophagy in wild type and Atg5-/- mouse embryonic fibroblasts: a survival mechanism in nonmalignant cells. 42nd Congress of the Federation-of-European-Biochemical-Societies (FEBS) on From Molecules to Cells and Back , (Jerusalem, Israel), 307.

Adacan, K., YERLİKAYA, P. O., Arisan, E. D., ÇOKER GÜRKAN, A., & Unsal, N. P., (2017). Endoplasmic reticulum stress is activated after epibrassinolide treatment leading autophagy in wild type and Atg5-/- mouse embryonic fibroblasts: a survival mechanism in nonmalignant cells . 42nd Congress of the Federation-of-European-Biochemical-Societies (FEBS) on From Molecules to Cells and Back (pp.307). Jerusalem, Israel

Adacan, K. Et Al. "Endoplasmic reticulum stress is activated after epibrassinolide treatment leading autophagy in wild type and Atg5-/- mouse embryonic fibroblasts: a survival mechanism in nonmalignant cells," 42nd Congress of the Federation-of-European-Biochemical-Societies (FEBS) on From Molecules to Cells and Back, Jerusalem, Israel, 2017

Adacan, K. Et Al. "Endoplasmic reticulum stress is activated after epibrassinolide treatment leading autophagy in wild type and Atg5-/- mouse embryonic fibroblasts: a survival mechanism in nonmalignant cells." 42nd Congress of the Federation-of-European-Biochemical-Societies (FEBS) on From Molecules to Cells and Back , Jerusalem, Israel, pp.307, 2017

Adacan, K. Et Al. (2017) . "Endoplasmic reticulum stress is activated after epibrassinolide treatment leading autophagy in wild type and Atg5-/- mouse embryonic fibroblasts: a survival mechanism in nonmalignant cells." 42nd Congress of the Federation-of-European-Biochemical-Societies (FEBS) on From Molecules to Cells and Back , Jerusalem, Israel, p.307.

@conferencepaper{conferencepaper, author={K. Adacan Et Al. }, title={Endoplasmic reticulum stress is activated after epibrassinolide treatment leading autophagy in wild type and Atg5-/- mouse embryonic fibroblasts: a survival mechanism in nonmalignant cells}, congress name={42nd Congress of the Federation-of-European-Biochemical-Societies (FEBS) on From Molecules to Cells and Back}, city={Jerusalem}, country={Israel}, year={2017}, pages={307} }