S. Aras Et Al. , "Orlistat as a fatty acid synthase inhibitor blocks cell survival mechanism via activating PTEN, which controls PI3K/AKT/MAPK axis in PC3 prostate cancer cells," European Biotechnology Congress , Dubrovnik, Croatia, 2017
Aras, S. Et Al. 2017. Orlistat as a fatty acid synthase inhibitor blocks cell survival mechanism via activating PTEN, which controls PI3K/AKT/MAPK axis in PC3 prostate cancer cells. European Biotechnology Congress , (Dubrovnik, Croatia).
Aras, S., Ozdemir, B. S., Arisan, E. D., RENCÜZOĞULLARI, Ö., ÖZFİLİZ KILBAŞ, P., YERLİKAYA, P. O., ... ÇOKER GÜRKAN, A.(2017). Orlistat as a fatty acid synthase inhibitor blocks cell survival mechanism via activating PTEN, which controls PI3K/AKT/MAPK axis in PC3 prostate cancer cells . European Biotechnology Congress, Dubrovnik, Croatia
Aras, Sahra Et Al. "Orlistat as a fatty acid synthase inhibitor blocks cell survival mechanism via activating PTEN, which controls PI3K/AKT/MAPK axis in PC3 prostate cancer cells," European Biotechnology Congress, Dubrovnik, Croatia, 2017
Aras, Sahra Et Al. "Orlistat as a fatty acid synthase inhibitor blocks cell survival mechanism via activating PTEN, which controls PI3K/AKT/MAPK axis in PC3 prostate cancer cells." European Biotechnology Congress , Dubrovnik, Croatia, 2017
Aras, S. Et Al. (2017) . "Orlistat as a fatty acid synthase inhibitor blocks cell survival mechanism via activating PTEN, which controls PI3K/AKT/MAPK axis in PC3 prostate cancer cells." European Biotechnology Congress , Dubrovnik, Croatia.
@conferencepaper{conferencepaper, author={Sahra Aras Et Al. }, title={Orlistat as a fatty acid synthase inhibitor blocks cell survival mechanism via activating PTEN, which controls PI3K/AKT/MAPK axis in PC3 prostate cancer cells}, congress name={European Biotechnology Congress}, city={Dubrovnik}, country={Croatia}, year={2017}}